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Published Erratum
. 2025 Jan 31;15(1):21.
doi: 10.1186/s13613-024-01403-1.

Correction: Fluid accumulation syndrome in sepsis and septic shock: pathophysiology, relevance and treatment-a comprehensive review

Affiliations
Published Erratum

Correction: Fluid accumulation syndrome in sepsis and septic shock: pathophysiology, relevance and treatment-a comprehensive review

Carmen Andrea Pfortmueller et al. Ann Intensive Care. .
No abstract available

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Figures

Fig. 1
Fig. 1
Critical appraisal of fluid accumulation syndrome (FAS)
Fig. 1
Fig. 1
Potential adverse consequences of fluid accumulation. Adapted with permission from Malbrain et al. according to the Open Access CC BY Licence 4.0 [3, 7, 8]. Effects mentioned are related to the setting of sepsis, capillary leak and fluid accumulation. I.e. the numbness refers to the presence of peripheral edema and anasarca that may cause skin conduction disturbances, compression of nerves, reduced blood flow and reduced mobility. Additionally, severe and prolonged fluid imbalances can lead to a range of health issues and complications, including electrolyte imbalances, which may indirectly affect the body's ability to respond to stress, including the production of cortisol by the adrenal glands. APP abdominal perfusion pressure (MAP minus IAP), RSB rapid shallow breathing, HCS hepatic congestion, GRV gastro-esophageal reflux, CARS cardiac-renal syndrome, AKI acute kidney injury, JVP jugular venous pressure, HJR hepato-jugular reflux
Fig. 2
Fig. 2
Potential adverse consequences of fluid accumulation. Adapted with permission from Malbrain et al. according to the Open Access CC BY Licence 4.0 [3, 7, 8]. Effects mentioned are related to the setting of sepsis, capillary leak and fluid accumulation. I.e. the numbness refers to the presence of peripheral edema and anasarca that may cause skin conduction disturbances, compression of nerves, reduced blood flow and reduced mobility. Additionally, severe and prolonged fluid imbalances can lead to a range of health issues and complications, including electrolyte imbalances, which may indirectly affect the body's ability to respond to stress, including the production of cortisol by the adrenal glands. APP abdominal perfusion pressure (MAP minus IAP), RSB rapid shallow breathing, HCS hepatic congestion, GRV gastro-esophageal reflux, CARS cardiac-renal syndrome, AKI acute kidney injury, JVP jugular venous pressure, HJR hepato-jugular reflux
Fig. 2
Fig. 2
The 4 phases conceptual ROSE model and deleterious effects of fluid accumulation syndrome. Adapted with permission from Malbrain et al. according to the Open Access CC BY Licence 4.0 [3, 7, 8]. IAP: intra-abdominal pressure, BIA: bio-impedance analysis, COP colloid oncotic pressure, ECW/ICW extracellular/intracellular water, EGDT early goal directed therapy, EVLWI extra-vascular lung water index, FAS fluid accumulation syndrome, FB fluid balance, GEDVI global end-diastolic volume index, IVCCI inferior vena cava collapsibility index, LVEDAI left ventricular end-diastolic area index, MAP mean arterial pressure, OCS ocular compartment syndrome, PAOP pulmonary artery occlusion pressure, PLR passive leg raising, PPV pulse pressure variation, PVPI pulmonary vascular permeability index, RVEDVI right ventricular end-diastolic volume index, RVR renal vascular resistance, ScvO2 central venous oxygen saturation, SvO2 mixed venous oxygen saturation, SV stroke volume, SVV stroke volume variation. Artwork kindly provided by Dr Ricardo Castro, Pontificia Universidad Católica de Chile, Chile

Erratum for

References

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