Comment on: "Switching TKIs during CML therapy is frequent, mostly driven by intolerance, and does not affect survival: a prospective Quebec registry study" by Busque et al
- PMID: 40251176
- PMCID: PMC12008287
- DOI: 10.1038/s41408-025-01288-8
Comment on: "Switching TKIs during CML therapy is frequent, mostly driven by intolerance, and does not affect survival: a prospective Quebec registry study" by Busque et al
Conflict of interest statement
Competing interests: The author declares no competing interests.
Comment on
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Switching TKIs during CML therapy is frequent, mostly driven by intolerance, and does not affect survival: a prospective Quebec registry study.Blood Cancer J. 2025 Mar 11;15(1):35. doi: 10.1038/s41408-025-01242-8. Blood Cancer J. 2025. PMID: 40069191 Free PMC article.
References
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- Hehlmann R, Cortes JE, Zyczynski T, Gambacorti-Passerini C, Goldberg SL, Mauro MJ, et al. Tyrosine kinase inhibitor interruptions, discontinuations and switching in patients with chronic-phase chronic myeloid leukemia in routine clinical practice: SIMPLICITY. Am J Hematol. 2019;94:46–54. - PMC - PubMed
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- Milojkovic D, Cross NCP, Ali S, Byrne J, Campbell G, Dignan FL, et al. Real-world tyrosine kinase inhibitor treatment pathways, monitoring patterns and responses in patients with chronic myeloid leukaemia in the United Kingdom: the UK TARGET CML study. Br J Haematol. 2021;192:62–74. - PubMed
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