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Published Erratum
. 2025 Apr 18;22(4):635.
doi: 10.3390/ijerph22040635.

Correction: Karoui et al. Nitrogen Dioxide Inhalation Exposures Induce Cardiac Mitochondrial Reactive Oxygen Species Production, Impair Mitochondrial Function and Promote Coronary Endothelial Dysfunction. Int. J. Environ. Res. Public Health 2020, 17, 5526

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Published Erratum

Correction: Karoui et al. Nitrogen Dioxide Inhalation Exposures Induce Cardiac Mitochondrial Reactive Oxygen Species Production, Impair Mitochondrial Function and Promote Coronary Endothelial Dysfunction. Int. J. Environ. Res. Public Health 2020, 17, 5526

Ahmed Karoui et al. Int J Environ Res Public Health. .

Abstract

In the original publication [...].

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Figures

Figure 5
Figure 5
Echocardiographic parameters after repeated NO2 exposures. Echocardiographic assessments were performed at 1-day post-exposure after 3 weeks (3 h/day, 5 days/week) of NO2 exposures. The diagram shows echocardiographic measurements of left ventricle (LV) end-diastolic diameter (A), LV end-systolic diameter (B), LV fractional shortening (C) and cardiac output (D). * p < 0.05, ** p < 0.01, *** p < 0.001, vs. control.

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References

    1. Karoui A., Crochemore C., Harouki N., Corbière C., Preterre D., Vendeville C., Richard V., Fardel O., Lecureur V., Vaugeois J.-M., et al. Nitrogen Dioxide Inhalation Exposures Induce Cardiac Mitochondrial Reactive Oxygen Species Production, Impair Mitochondrial Function and Promote Coronary Endothelial Dysfunction. Int. J. Environ. Res. Public Health. 2020;17:5526. doi: 10.3390/ijerph17155526. - DOI - PMC - PubMed

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