Cited by

• Acute lead poisoning in inherited porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency.

  Doss M, Müller WA. Doss M, et al. Blut. 1982 Aug;45(2):131-9. doi: 10.1007/BF00319941. Blut. 1982. PMID: 7104498
• Tissue distribution and urinary excretion of essential elements in rats orally exposed to aluminum chloride.

  Chmielnicka J, Nasiadek M. Chmielnicka J, et al. Biol Trace Elem Res. 1991 Nov;31(2):131-8. doi: 10.1007/BF02990421. Biol Trace Elem Res. 1991. PMID: 9438034
• Persistent protoporphyrinemia in hereditary porphobilinogen synthase (delta-aminolevulinic acid dehydrase) deficiency under low lead exposure. A new molecular basis for the pathogenesis of lead intoxication.

  Doss M, Becker U, Sixel F, Geisse S, Solcher H, Schneider J, Kufner G, Schlegel H, Stoeppler M. Doss M, et al. Klin Wochenschr. 1982 Jun 15;60(12):599-606. doi: 10.1007/BF01711435. Klin Wochenschr. 1982. PMID: 7109500
• Pre-Ride Biomarkers and Endurance Horse Welfare: Analyzing the Impact of the Elimination of Superoxide Dismutase, δ-Aminolevulinic-Dehydratase, Thiobarbituric Acid Reactive Substances, Iron, and Serum Amyloid A Levels in Elite 160 km Endurance Rides.

  Bollinger L, Bartel A, Weber C, Gehlen H. Bollinger L, et al. Animals (Basel). 2023 May 17;13(10):1670. doi: 10.3390/ani13101670. Animals (Basel). 2023. PMID: 37238102 Free PMC article.
• Evaluation of lead exposure in workers at a lead-acid battery factory in Korea: with focus on activity of erythrocyte pyrimidine 5'-nucleotidase (P5N).

  Kim Y, Harada K, Ohmori S, Lee BK, Miura H, Ueda A. Kim Y, et al. Occup Environ Med. 1995 Jul;52(7):484-8. doi: 10.1136/oem.52.7.484. Occup Environ Med. 1995. PMID: 7670624 Free PMC article.