Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
Comparative Study
. 1982 Feb;35(2):654-9.
doi: 10.1128/iai.35.2.654-659.1982.

Further characterization of complement resistance conferred on Escherichia coli by the plasmid genes traT of R100 and iss of ColV,I-K94

M M Binns, J Mayden, R P Levine
Comparative Study

Further characterization of complement resistance conferred on Escherichia coli by the plasmid genes traT of R100 and iss of ColV,I-K94

M M Binns et al. Infect Immun. 1982 Feb.

Abstract

We have shown that the traT gene product was responsible for the complement resistance of the R100 plasmid. We compared this resistance with that specified by the iss gene of the ColV,I-K94 plasmid. The levels of resistance specified by the two genes were similar, and there was no additive effect on resistance when both genes were present together. Under conditions in which traT and iss conferred at least a 50- and 10-fold increase in survival, respectively, the consumption of C6, C7, C8, and C9 was the same for bacteria with and without the plasmid genes. This result indicated that it was the action of the terminal complex, not its formation, which was blocked by traT and iss.

PubMed Disclaimer

References

    1. J Mol Biol. 1966 Mar;16(1):118-33 - PubMed
    1. Biochim Biophys Acta. 1972 Feb 14;265(1):1-23 - PubMed
    1. Proc Natl Acad Sci U S A. 1972 Aug;69(8):2110-4 - PubMed
    1. J Mol Biol. 1973 Apr 5;75(2):235-55 - PubMed
    1. Infection. 1973;1(1):24-8 - PubMed

Publication types

Substances