Symptomatic or secondary basal ganglia diseases and tardive dyskinesias
- PMID: 7582050
Symptomatic or secondary basal ganglia diseases and tardive dyskinesias
Abstract
This paper reviews clinical reports of various movement disorders (dystonia, chorea, ballism and parkinsonism) caused by well defined lesions of the putamen, globus pallidus, substantia nigra, thalamus and subthalamus. The role of basal ganglia in motor control is also emphasized.
Similar articles
-
The neuronal mechanism underlying parkinsonism and dyskinesia, and differential roles of the putamen and caudate nucleus.Adv Neurol. 1993;60:71-7. Adv Neurol. 1993. PMID: 8380531
-
Origin of chorea-ballism and multisystem degeneration: pathophysiological implications.Mov Disord. 1993;8(1):123-4. doi: 10.1002/mds.870080126. Mov Disord. 1993. PMID: 8419800 No abstract available.
-
[Somatotopy in the basal ganglia].Brain Nerve. 2009 Dec;61(12):1383-94. Brain Nerve. 2009. PMID: 20034305 Review. Japanese.
-
Dynamic aspects of the striatothalamic connection studied in cases with movement disorder.Adv Neurol. 1993;60:78-83. Adv Neurol. 1993. PMID: 8420220 No abstract available.
-
[Movement disorders and basal ganglia function].Tidsskr Nor Laegeforen. 2008 Sep 11;128(17):1968-71. Tidsskr Nor Laegeforen. 2008. PMID: 18787574 Review. Norwegian.
Cited by
-
Variations of movement disorders in anti-N-methyl-D-aspartate receptor encephalitis: A nationwide study in Taiwan.Medicine (Baltimore). 2016 Sep;95(37):e4365. doi: 10.1097/MD.0000000000004365. Medicine (Baltimore). 2016. PMID: 27631202 Free PMC article.