p53: a cell cycle regulator activated by DNA damage
- PMID: 7793313
- DOI: 10.1016/s0065-230x(08)60253-5
p53: a cell cycle regulator activated by DNA damage
Similar articles
-
[TP53 tumor suppressor gene: 20 years (and ten thousand mutations) later].Bull Cancer. 2000 Jan;87(1):11-8. Bull Cancer. 2000. PMID: 10673626 French. No abstract available.
-
[Tumor suppressor gene p53].Nihon Rinsho. 1996 Apr;54(4):960-4. Nihon Rinsho. 1996. PMID: 8920657 Review. Japanese.
-
The molecular perspective: p53 tumor suppressor.Oncologist. 1999;4(2):138-9. Oncologist. 1999. PMID: 10337384 No abstract available.
-
The role of p53-target genes in human cancer.Crit Rev Oncol Hematol. 2000 Jan;33(1):1-6. doi: 10.1016/s1040-8428(99)00051-7. Crit Rev Oncol Hematol. 2000. PMID: 10714958 Review.
-
Mutations in the p53 tumor suppressor gene: clues to cancer etiology and molecular pathogenesis.Cancer Res. 1994 Sep 15;54(18):4855-78. Cancer Res. 1994. PMID: 8069852 Review. No abstract available.
Cited by
-
New p53-based anti-cancer therapeutic strategies.Med Oncol. 1998 Dec;15(4):222-8. doi: 10.1007/BF02787204. Med Oncol. 1998. PMID: 9951684 Review.
-
P53 mutations associated with breast, colorectal, liver, lung, and ovarian cancers.Environ Health Perspect. 1996 Dec;104(12):1324-31. doi: 10.1289/ehp.961041324. Environ Health Perspect. 1996. PMID: 9118874 Free PMC article.
-
T cells from baxalpha transgenic mice show accelerated apoptosis in response to stimuli but do not show restored DNA damage-induced cell death in the absence of p53.EMBO J. 1996 Mar 15;15(6):1221-30. EMBO J. 1996. PMID: 8635454 Free PMC article.
-
CD3-epsilon overexpressed in prothymocytes acts as an oncogene.Mol Med. 1997 Jan;3(1):72-81. Mol Med. 1997. PMID: 9132282 Free PMC article.
-
Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance.Proc Natl Acad Sci U S A. 1995 Sep 12;92(19):8876-80. doi: 10.1073/pnas.92.19.8876. Proc Natl Acad Sci U S A. 1995. PMID: 7568035 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Research Materials
Miscellaneous