Similar articles

• Transmission barriers for bovine, ovine, and human prions in transgenic mice.

  Scott MR, Peretz D, Nguyen HO, Dearmond SJ, Prusiner SB. Scott MR, et al. J Virol. 2005 May;79(9):5259-71. doi: 10.1128/JVI.79.9.5259-5271.2005. J Virol. 2005. PMID: 15827140 Free PMC article.
• Insights into Mechanisms of Transmission and Pathogenesis from Transgenic Mouse Models of Prion Diseases.

  Moreno JA, Telling GC. Moreno JA, et al. Methods Mol Biol. 2017;1658:219-252. doi: 10.1007/978-1-4939-7244-9_16. Methods Mol Biol. 2017. PMID: 28861793 Free PMC article. Review.
• Increased expression of water channel aquaporin 1 and aquaporin 4 in Creutzfeldt-Jakob disease and in bovine spongiform encephalopathy-infected bovine-PrP transgenic mice.

  Rodríguez A, Pérez-Gracia E, Espinosa JC, Pumarola M, Torres JM, Ferrer I. Rodríguez A, et al. Acta Neuropathol. 2006 Nov;112(5):573-85. doi: 10.1007/s00401-006-0117-1. Epub 2006 Jul 27. Acta Neuropathol. 2006. PMID: 16871401
• Differences in proteinase K resistance and neuronal deposition of abnormal prion proteins characterize bovine spongiform encephalopathy (BSE) and scrapie strains.

  Kuczius T, Groschup MH. Kuczius T, et al. Mol Med. 1999 Jun;5(6):406-18. Mol Med. 1999. PMID: 10415165 Free PMC article.
• Bovine spongiform encephalopathy--some surprises for biochemists.

  Campbell PN. Campbell PN. IUBMB Life. 2005 Apr-May;57(4-5):273-6. doi: 10.1080/15216540500097152. IUBMB Life. 2005. PMID: 16036610 Review.