Review
doi: 10.1073/pnas.90.14.6381.
Alzheimer disease and the prion disorders amyloid beta-protein and prion protein amyloidoses
Affiliations
- PMID: 8101988
- PMCID: PMC46935
- DOI: 10.1073/pnas.90.14.6381
Item in Clipboard
Review
Alzheimer disease and the prion disorders amyloid beta-protein and prion protein amyloidoses
Proc Natl Acad Sci U S A.
.
Erratum in
- Proc Natl Acad Sci U S A 1993 Oct 1;90(19):9233
Abstract
Alzheimer disease and the prion disorders/spongiform encephalopathies share many common features. These chronic, progressive, sometimes familial diseases of the central nervous system are characterized by the presence of different types of amyloid deposits in the brain. This review provides a perspective on these two types of neurodegenerative disorders.
Similar articles
-
Application of yeast to studying amyloid and prion diseases.Adv Genet. 2020;105:293-380. doi: 10.1016/bs.adgen.2020.01.002. Epub 2020 May 4. Adv Genet. 2020. PMID: 32560789 Free PMC article. Review.
-
[Amyloidgenesis in prion disease].Brain Nerve. 2014 Jul;66(7):849-55. Brain Nerve. 2014. PMID: 24998829 Japanese.
-
Pathogenic mechanisms of prion protein, amyloid-β and α-synuclein misfolding: the prion concept and neurotoxicity of protein oligomers.J Neurochem. 2016 Oct;139(2):162-180. doi: 10.1111/jnc.13772. Epub 2016 Sep 15. J Neurochem. 2016. PMID: 27529376 Review.
-
Neurotoxicity of beta-amyloid and prion peptides.Curr Opin Neurol. 1996 Dec;9(6):492-500. doi: 10.1097/00019052-199612000-00017. Curr Opin Neurol. 1996. PMID: 9007411 Review.
-
Transmissible spongiform encephalopathies: virally transmitted amyloidoses.Dev Biol Stand. 1993;80:25-9. Dev Biol Stand. 1993. PMID: 8270112 Review. No abstract available.
Cited by
-
Genetic variability of the gene cluster CALHM 1-3 in sporadic Creutzfeldt-Jakob disease.Prion. 2012 Sep-Oct;6(4):407-12. doi: 10.4161/pri.20785. Epub 2012 Aug 9. Prion. 2012. PMID: 22874670 Free PMC article.
-
Ribozyme mediated degradation of beta-amyloid peptide precursor mRNA in COS-7 cells.Nucleic Acids Res. 1994 Jun 25;22(12):2375-82. doi: 10.1093/nar/22.12.2375. Nucleic Acids Res. 1994. PMID: 8036167 Free PMC article.
-
Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell death.Proc Natl Acad Sci U S A. 1994 Feb 15;91(4):1470-4. doi: 10.1073/pnas.91.4.1470. Proc Natl Acad Sci U S A. 1994. PMID: 8108433 Free PMC article.
-
Lipid thioesters derived from acylated proteins accumulate in infantile neuronal ceroid lipofuscinosis: correction of the defect in lymphoblasts by recombinant palmitoyl-protein thioesterase.Proc Natl Acad Sci U S A. 1996 Sep 17;93(19):10046-50. doi: 10.1073/pnas.93.19.10046. Proc Natl Acad Sci U S A. 1996. PMID: 8816748 Free PMC article.
-
A common BACE1 polymorphism is a risk factor for sporadic Creutzfeldt-Jakob disease.PLoS One. 2012;7(8):e43926. doi: 10.1371/journal.pone.0043926. Epub 2012 Aug 30. PLoS One. 2012. PMID: 22952813 Free PMC article.
References
Publication types
MeSH terms
Substances
Grants and funding
LinkOut - more resources
Full Text Sources
Other Literature Sources
Medical
