Is NO an endogenous antiatherogenic molecule?

Editorial

J P Cooke et al. Arterioscler Thromb. 1994 May.

. 1994 May;14(5):653-5.

doi: 10.1161/01.atv.14.5.653.

No abstract available

Comment on

Chronic inhibition of nitric oxide production accelerates neointima formation and impairs endothelial function in hypercholesterolemic rabbits.
Cayatte AJ, Palacino JJ, Horten K, Cohen RA. Cayatte AJ, et al. Arterioscler Thromb. 1994 May;14(5):753-9. doi: 10.1161/01.atv.14.5.753. Arterioscler Thromb. 1994. PMID: 7513551
Long-term inhibition of NO synthesis promotes atherosclerosis in the hypercholesterolemic rabbit thoracic aorta. PGH2 does not contribute to impaired endothelium-dependent relaxation.
Naruse K, Shimizu K, Muramatsu M, Toki Y, Miyazaki Y, Okumura K, Hashimoto H, Ito T. Naruse K, et al. Arterioscler Thromb. 1994 May;14(5):746-52. doi: 10.1161/01.atv.14.5.746. Arterioscler Thromb. 1994. PMID: 8172852