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Clinical Trial
. 1997 Oct;65(5):715-26.
doi: 10.1037//0022-006x.65.5.715.

Cognitive-behavioral treatment for depression in alcoholism

Affiliations
Clinical Trial

Cognitive-behavioral treatment for depression in alcoholism

R A Brown et al. J Consult Clin Psychol. 1997 Oct.

Abstract

Alcoholics with depressive symptoms score > or = 10 on the Beck Depression Inventory (A.T. Beck, C. H. Ward, M. Mendelson, J. Mock, & J. Erbaugh, 1961) received 8 individual sessions of cognitive-behavioral treatment for depression (CBT-D, n = 19) or a relaxation training control (RTC; n = 16) plus standard alcohol treatment. CBT-D patients had greater reductions in somatic depressive symptoms and depressed and anxious mood than RTC patients during treatment. Patients receiving CBT-D had a greater percentage of days abstinent but not greater overall abstinence or fewer drinks per day during the first 3-month follow-up. However, between the 3- and 6-month follow-ups, CBT-D patients had significantly better alcohol use outcomes on total abstinence (47% vs. 13%), percent days abstinent (90.5% vs. 68.3%), and drinks per day (0.46 vs. 5.71). Theoretical and clinical implications of using CBT-D in alcohol treatment are discussed.

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Figures

Figure 1
Figure 1
Depressive symptoms and negative moods by treatment condition and time. CBT-D = cognitive-behavioral treatment for depression; RTC = relaxation training control; HAM-D = Modified Hamilton Rating Scale for Depression; POMS = Profile of Mood States; Depression = Depression subscale; Anxiety = Anxiety subscale.
Figure 2
Figure 2
Drinking frequency and quantity at follow-up by treatment condition and time. CBT-D cognitive–behavioral treatment for depression; RTC = relaxation training control.
Figure 3
Figure 3
The mediating effect of change in depressive symptoms on the relationship between treatment and drinking outcome. A series of regression analyses were performed with the appropriate baseline drinking variable controlled for by entering it first in all regression analyses. First, change in depressive symptoms was regressed onto treatment condition (A). Second, drinking outcome was regressed onto change in depressive symptoms (B). Third, drinking outcome was regressed onto treatment condition (C). Fourth, drinking outcome was regressed onto treatment condition after controlling for change in depressive symptoms (D). Numbers represent squared correlations, z = the decrease from .22 to .14 approached significance, p < .08. HAM-D = Modified Hamilton Rating Scale for Depression. *p < .05.

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