Similar articles

• Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.

  Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD. Rusyn EV, et al. Oncogene. 2000 Sep 28;19(41):4685-94. doi: 10.1038/sj.onc.1203836. Oncogene. 2000. PMID: 11032018
• Regulation of cell cycle progression and apoptosis by the Ras/Raf/MEK/ERK pathway (Review).

  Chang F, Steelman LS, Shelton JG, Lee JT, Navolanic PM, Blalock WL, Franklin R, McCubrey JA. Chang F, et al. Int J Oncol. 2003 Mar;22(3):469-80. Int J Oncol. 2003. PMID: 12579299 Review.
• [Mechanism of interaction of ras with its effectors].

  Kariya K, Kataoka T. Kariya K, et al. Seikagaku. 2000 Apr;72(4):285-8. Seikagaku. 2000. PMID: 10853365 Review. Japanese. No abstract available.
• The Raf signal transduction cascade as a target for chemotherapeutic intervention in growth factor-responsive tumors.

  Weinstein-Oppenheimer CR, Blalock WL, Steelman LS, Chang F, McCubrey JA. Weinstein-Oppenheimer CR, et al. Pharmacol Ther. 2000 Dec;88(3):229-79. doi: 10.1016/s0163-7258(00)00085-1. Pharmacol Ther. 2000. PMID: 11337027 Review.
• K-Ras and H-Ras activation promote distinct consequences on endometrial cell survival.

  Ninomiya Y, Kato K, Takahashi A, Ueoka Y, Kamikihara T, Arima T, Matsuda T, Kato H, Nishida J, Wake N. Ninomiya Y, et al. Cancer Res. 2004 Apr 15;64(8):2759-65. doi: 10.1158/0008-5472.can-3487-2. Cancer Res. 2004. PMID: 15087391