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Destabilization of Na(v)1.7 sodium channel alpha-subunit mRNA by constitutive phosphorylation of extracellular signal-regulated kinase: negative regulation of steady-state level of cell surface functional sodium channels in adrenal chromaffin cells.
Yanagita T, Kobayashi H, Uezono Y, Yokoo H, Sugano T, Saitoh T, Minami S, Shiraishi S, Wada A. Yanagita T, et al. Among authors: uezono y. Mol Pharmacol. 2003 May;63(5):1125-36. doi: 10.1124/mol.63.5.1125. Mol Pharmacol. 2003. PMID: 12695541
Lysophosphatidic acid-LPA1 receptor-Rho-Rho kinase-induced up-regulation of Nav1.7 sodium channel mRNA and protein in adrenal chromaffin cells: enhancement of 22Na+ influx, 45Ca2+ influx and catecholamine secretion.
Maruta T, Yanagita T, Matsuo K, Uezono Y, Satoh S, Nemoto T, Yoshikawa N, Kobayashi H, Takasaki M, Wada A. Maruta T, et al. Among authors: uezono y. J Neurochem. 2008 Apr;105(2):401-12. doi: 10.1111/j.1471-4159.2007.05143.x. Epub 2007 Nov 25. J Neurochem. 2008. PMID: 18036192 Free article.
Chronic lithium treatment up-regulates cell surface Na(V)1.7 sodium channels via inhibition of glycogen synthase kinase-3 in adrenal chromaffin cells: enhancement of Na(+) influx, Ca(2+) influx and catecholamine secretion after lithium withdrawal.
Yanagita T, Maruta T, Nemoto T, Uezono Y, Matsuo K, Satoh S, Yoshikawa N, Kanai T, Kobayashi H, Wada A. Yanagita T, et al. Among authors: uezono y. Neuropharmacology. 2009 Sep;57(3):311-21. doi: 10.1016/j.neuropharm.2009.05.006. Epub 2009 May 30. Neuropharmacology. 2009. PMID: 19486905
256 results