Block T - Search Results

1

A review of the molecular mechanism of HSV-1 latency.

Fraser NW, Spivack JG, Wroblewska Z, Block T, Deshmane SL, Valyi-Nagy T, Natarajan R, Gesser RM. Fraser NW, et al. Among authors: block t. Curr Eye Res. 1991;10 Suppl:1-13. doi: 10.3109/02713689109020352. Curr Eye Res. 1991. PMID: 1650659 Review.

2

An HSV-1 containing the rat beta-glucuronidase cDNA inserted within the LAT gene is less efficient than the parental strain at establishing a transcriptionally active state during latency in neurons.

Deshmane SL, Valyi-Nagy T, Block T, Maggioncalda J, Wolfe JH, Dillner A, Fraser NW. Deshmane SL, et al. Among authors: block t. Gene Ther. 1995 May;2(3):209-17. Gene Ther. 1995. PMID: 7614252

3

The latency-associated transcripts of herpes simplex virus: RNA in search of function.

Fraser NW, Block TM, Spivack JG. Fraser NW, et al. Among authors: block tm. Virology. 1992 Nov;191(1):1-8. doi: 10.1016/0042-6822(92)90160-q. Virology. 1992. PMID: 1329311 Review. No abstract available.

4

A herpes simplex virus type 1 latency-associated transcript mutant reactivates with normal kinetics from latent infection.

Block TM, Spivack JG, Steiner I, Deshmane S, McIntosh MT, Lirette RP, Fraser NW. Block TM, et al. J Virol. 1990 Jul;64(7):3417-26. doi: 10.1128/JVI.64.7.3417-3426.1990. J Virol. 1990. PMID: 2161947 Free PMC article.

5

An HSV LAT null mutant reactivates slowly from latent infection and makes small plaques on CV-1 monolayers.

Block TM, Deshmane S, Masonis J, Maggioncalda J, Valyi-Nagi T, Fraser NW. Block TM, et al. Virology. 1993 Feb;192(2):618-30. doi: 10.1006/viro.1993.1078. Virology. 1993. PMID: 8380666

6

Long term herpes simplex virus type 1 infection of nerve growth factor-treated PC12 cells.

Block T, Barney S, Masonis J, Maggioncalda J, Valyi-Nagy T, Fraser NW. Block T, et al. J Gen Virol. 1994 Sep;75 ( Pt 9):2481-7. doi: 10.1099/0022-1317-75-9-2481. J Gen Virol. 1994. PMID: 8077952

7

In situ DNA PCR and RNA hybridization detection of herpes simplex virus sequences in trigeminal ganglia of latently infected mice.

Mehta A, Maggioncalda J, Bagasra O, Thikkavarapu S, Saikumari P, Valyi-Nagy T, Fraser NW, Block TM. Mehta A, et al. Among authors: block tm. Virology. 1995 Jan 10;206(1):633-40. doi: 10.1016/s0042-6822(95)80080-8. Virology. 1995. PMID: 7831818 Free article.

8

Analysis of a herpes simplex virus type 1 LAT mutant with a deletion between the putative promoter and the 5' end of the 2.0-kilobase transcript.

Maggioncalda J, Mehta A, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: block tm. J Virol. 1994 Dec;68(12):7816-24. doi: 10.1128/JVI.68.12.7816-7824.1994. J Virol. 1994. PMID: 7966571 Free PMC article.

9

A herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics.

Maggioncalda J, Mehta A, Bagasra O, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: block tm. J Neurovirol. 1996 Aug;2(4):268-78. doi: 10.3109/13550289609146890. J Neurovirol. 1996. PMID: 8799218

10

Correlation between herpes simplex virus type 1 rate of reactivation from latent infection and the number of infected neurons in trigeminal ganglia.

Maggioncalda J, Mehta A, Su YH, Fraser NW, Block TM. Maggioncalda J, et al. Among authors: block tm. Virology. 1996 Nov 1;225(1):72-81. doi: 10.1006/viro.1996.0576. Virology. 1996. PMID: 8918535 Free article.

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