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Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channels.
Lübkemeier I, Requardt RP, Lin X, Sasse P, Andrié R, Schrickel JW, Chkourko H, Bukauskas FF, Kim JS, Frank M, Malan D, Zhang J, Wirth A, Dobrowolski R, Mohler PJ, Offermanns S, Fleischmann BK, Delmar M, Willecke K. Lübkemeier I, et al. Among authors: kim js. Basic Res Cardiol. 2013 May;108(3):348. doi: 10.1007/s00395-013-0348-y. Epub 2013 Apr 5. Basic Res Cardiol. 2013. PMID: 23558439 Free PMC article.
The conditional connexin43G138R mouse mutant represents a new model of hereditary oculodentodigital dysplasia in humans.
Dobrowolski R, Sasse P, Schrickel JW, Watkins M, Kim JS, Rackauskas M, Troatz C, Ghanem A, Tiemann K, Degen J, Bukauskas FF, Civitelli R, Lewalter T, Fleischmann BK, Willecke K. Dobrowolski R, et al. Among authors: kim js. Hum Mol Genet. 2008 Feb 15;17(4):539-54. doi: 10.1093/hmg/ddm329. Epub 2007 Nov 13. Hum Mol Genet. 2008. PMID: 18003637 Free PMC article.
Cardiomyocyte-restricted deletion of connexin43 during mouse development.
Eckardt D, Kirchhoff S, Kim JS, Degen J, Theis M, Ott T, Wiesmann F, Doevendans PA, Lamers WH, de Bakker JM, van Rijen HV, Schneider MD, Willecke K. Eckardt D, et al. Among authors: kim js. J Mol Cell Cardiol. 2006 Dec;41(6):963-71. doi: 10.1016/j.yjmcc.2006.07.017. Epub 2006 Sep 11. J Mol Cell Cardiol. 2006. PMID: 16963078
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