Perl A - Search Results

1

Mechanistic target of rapamycin complex 1 expands Th17 and IL-4+ CD4-CD8- double-negative T cells and contracts regulatory T cells in systemic lupus erythematosus.

Kato H, Perl A. Kato H, et al. Among authors: perl a. J Immunol. 2014 May 1;192(9):4134-44. doi: 10.4049/jimmunol.1301859. Epub 2014 Mar 28. J Immunol. 2014. PMID: 24683191 Free PMC article.

2

Elevation of mitochondrial transmembrane potential and reactive oxygen intermediate levels are early events and occur independently from activation of caspases in Fas signaling.

Banki K, Hutter E, Gonchoroff NJ, Perl A. Banki K, et al. Among authors: perl a. J Immunol. 1999 Feb 1;162(3):1466-79. J Immunol. 1999. PMID: 9973403 Free PMC article.

3

Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus.

Gergely P Jr, Grossman C, Niland B, Puskas F, Neupane H, Allam F, Banki K, Phillips PE, Perl A. Gergely P Jr, et al. Among authors: perl a. Arthritis Rheum. 2002 Jan;46(1):175-90. doi: 10.1002/1529-0131(200201)46:1<175::AID-ART10015>3.0.CO;2-H. Arthritis Rheum. 2002. PMID: 11817589 Free PMC article.

4

Persistent mitochondrial hyperpolarization, increased reactive oxygen intermediate production, and cytoplasmic alkalinization characterize altered IL-10 signaling in patients with systemic lupus erythematosus.

Gergely P Jr, Niland B, Gonchoroff N, Pullmann R Jr, Phillips PE, Perl A. Gergely P Jr, et al. Among authors: perl a. J Immunol. 2002 Jul 15;169(2):1092-101. doi: 10.4049/jimmunol.169.2.1092. J Immunol. 2002. PMID: 12097418 Free PMC article.

5

T cell activation-induced mitochondrial hyperpolarization is mediated by Ca2+- and redox-dependent production of nitric oxide.

Nagy G, Koncz A, Perl A. Nagy G, et al. Among authors: perl a. J Immunol. 2003 Nov 15;171(10):5188-97. doi: 10.4049/jimmunol.171.10.5188. J Immunol. 2003. PMID: 14607919 Free PMC article.

6

Mitochondrial dysfunction in T cells of patients with systemic lupus erythematosus.

Perl A, Gergely P Jr, Banki K. Perl A, et al. Int Rev Immunol. 2004 May-Aug;23(3-4):293-313. doi: 10.1080/08830180490452576. Int Rev Immunol. 2004. PMID: 15204090 Review.

7

Apoptosis and mitochondrial dysfunction in lymphocytes of patients with systemic lupus erythematosus.

Perl A, Nagy G, Gergely P, Puskas F, Qian Y, Banki K. Perl A, et al. Methods Mol Med. 2004;102:87-114. doi: 10.1385/1-59259-805-6:087. Methods Mol Med. 2004. PMID: 15286382

8

Nitric oxide-dependent mitochondrial biogenesis generates Ca2+ signaling profile of lupus T cells.

Nagy G, Barcza M, Gonchoroff N, Phillips PE, Perl A. Nagy G, et al. Among authors: perl a. J Immunol. 2004 Sep 15;173(6):3676-83. doi: 10.4049/jimmunol.173.6.3676. J Immunol. 2004. PMID: 15356113 Free PMC article.

9

T- and B-cell abnormalities in systemic lupus erythematosus.

Nagy G, Koncz A, Perl A. Nagy G, et al. Among authors: perl a. Crit Rev Immunol. 2005;25(2):123-40. doi: 10.1615/critrevimmunol.v25.i2.30. Crit Rev Immunol. 2005. PMID: 15952933 Review.

10

CD8+ T cell-mediated HLA-A*0201-restricted cytotoxicity to transaldolase peptide 168-176 in patients with multiple sclerosis.

Niland B, Banki K, Biddison WE, Perl A. Niland B, et al. Among authors: perl a. J Immunol. 2005 Dec 15;175(12):8365-78. doi: 10.4049/jimmunol.175.12.8365. J Immunol. 2005. PMID: 16339578

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