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Page 1
Did you mean borys (942 results)?
BORIS: a key regulator of cancer stemness.
Soltanian S, Dehghani H. Soltanian S, et al. Cancer Cell Int. 2018 Oct 5;18:154. doi: 10.1186/s12935-018-0650-8. eCollection 2018. Cancer Cell Int. 2018. PMID: 30323717 Free PMC article. Review.
BORIS (CTCFL) is a DNA binding protein which is involved in tumorigenesis. Although, there are different opinions on the level of gene expression and function of BORIS in normal and cancer tissues, the results of many studies have classified BORIS as a protei …
BORIS (CTCFL) is a DNA binding protein which is involved in tumorigenesis. Although, there are different opinions on the level of gen …
Targeting CTCFL/BORIS for the immunotherapy of cancer.
Loukinov D. Loukinov D. Cancer Immunol Immunother. 2018 Dec;67(12):1955-1965. doi: 10.1007/s00262-018-2251-8. Epub 2018 Nov 2. Cancer Immunol Immunother. 2018. PMID: 30390146 Free PMC article. Review.
Recently CTCFL/BORIS has received additional attention as a target expressed in cancer stem cells (CSC). These cells drive tumor growth recurrence, metastasis, and treatment resistance. CTCFL/BORIS silencing leads to senescence and death of CSC. Therefore, an immuno …
Recently CTCFL/BORIS has received additional attention as a target expressed in cancer stem cells (CSC). These cells drive tumor grow …
BORIS in human cancers -- a review.
Martin-Kleiner I. Martin-Kleiner I. Eur J Cancer. 2012 Apr;48(6):929-35. doi: 10.1016/j.ejca.2011.09.009. Epub 2011 Oct 21. Eur J Cancer. 2012. PMID: 22019212 Review.
Re-expression of BORIS in cancers is due to the hypomethylation of its promoter. High expression of BORIS protein and RNA correlates with the tumour size and grade in cancer patients. ...BORIS antitumor vaccines have been tested in mice with several cancers, …
Re-expression of BORIS in cancers is due to the hypomethylation of its promoter. High expression of BORIS protein and RNA corr …
CTCF and BORIS in genome regulation and cancer.
Marshall AD, Bailey CG, Rasko JE. Marshall AD, et al. Curr Opin Genet Dev. 2014 Feb;24:8-15. doi: 10.1016/j.gde.2013.10.011. Epub 2013 Dec 14. Curr Opin Genet Dev. 2014. PMID: 24657531 Review.
BORIS is aberrantly expressed frequently in cancer, leading some to propose a pro-tumourigenic role for BORIS. However, BORIS can inhibit cell proliferation, and is mutated in cancer similarly to CTCF suggesting BORIS activation in cancer may be due to …
BORIS is aberrantly expressed frequently in cancer, leading some to propose a pro-tumourigenic role for BORIS. However, BOR …
BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sites.
Pugacheva EM, Bhatt DN, Rivero-Hinojosa S, Tajmul M, Fedida L, Price E, Ji Y, Loukinov D, Strunnikov AV, Ren B, Lobanenkov VV. Pugacheva EM, et al. Genome Biol. 2024 Jan 31;25(1):40. doi: 10.1186/s13059-024-03175-0. Genome Biol. 2024. PMID: 38297316 Free PMC article.
We show that intergenic and intronic CTCF binding sites, which are transcriptionally inert in normal somatic cells, could be epigenetically reprogrammed into active de novo promoters in germ and cancer cells. BORIS/CTCFL, the testis-specific paralog of the ubiquitously exp …
We show that intergenic and intronic CTCF binding sites, which are transcriptionally inert in normal somatic cells, could be epigenetically …
BORIS-mediated generation of circular RNAs induces inflammation.
Xu H, Fang M, Li C, Zuo B, Ren J, Zhang Y. Xu H, et al. Transl Oncol. 2022 Apr;18:101363. doi: 10.1016/j.tranon.2022.101363. Epub 2022 Feb 16. Transl Oncol. 2022. PMID: 35182955 Free PMC article.
Brother of regulator of imprinting site (BORIS) promotes cancer progression and is specifically highly expressed in the majority of carcinoma. However, the mechanism underlying the regulation of circRNAs by the oncoprotein BORIS and their role in regulating inflamma …
Brother of regulator of imprinting site (BORIS) promotes cancer progression and is specifically highly expressed in the majority of c …
Boris Yegorov.
Kyle RA, Shampo MA. Kyle RA, et al. JAMA. 1972 Oct 9;222(2):177. doi: 10.1001/jama.222.2.177. JAMA. 1972. PMID: 4560298 No abstract available.
Boris knockout eliminates AOM/DSS-induced in situ colorectal cancer by suppressing DNA damage repair and inflammation.
Zuo BW, Yao WX, Fang MD, Ren J, Tu LL, Fan RJ, Zhang YM. Zuo BW, et al. Cancer Sci. 2023 May;114(5):1972-1985. doi: 10.1111/cas.15732. Epub 2023 Feb 5. Cancer Sci. 2023. PMID: 36692143 Free PMC article.
However, it is unknown whether Boris plays a role in the progression of in situ colorectal cancer. Here Boris knockout (KO) mice were constructed. ...Our data showed that Boris knockout alleviated DSS-induced colitis and that Boris knockdown inhibited …
However, it is unknown whether Boris plays a role in the progression of in situ colorectal cancer. Here Boris knockout (KO) mi …
4,067 results